Influencia de los herpesvirus en la patogénesis periapical

Autores/as

  • Andrés Herrera García Universidad Latina de Costa Rica
  • Sergio Castro Mora Universidad Latina de Costa Rica

DOI:

https://doi.org/10.59334/ROV.v1i22.285

Palabras clave:

Virus, herpes, periodontitis apical, coinfección

Resumen

La homeostasis de los tejidos periapicales se puede ver violentada por diferentes procesos como la periodontitis apical, y teniendo como causa más frecuente los agentes bacterianos. Técnicas diagnósticas como la inmunohistoquímica y la reacción en cadena de polimerasa (PCR), permiten un mejor entendimiento de la periodontitis apical. De esta manera es que se ha conseguido aislar indicios virales (transcripciones víricas) en lesiones periapicales, las cuales aparentemente tienen efectos sinérgicos con las bacterias. Esta revisión de literatura tiene por objetivo reforzar una nueva perspectiva de la periodontitis apical.

Descargas

Los datos de descargas todavía no están disponibles.

Citas

Andric, M., Milasin, J., Jovanovic, T., Todorovic, L., (2007). Human cytomegalovirus is present in odontogenic cysts. Oral Microbiology & Immunology, 22, 347–351. https://doi.org/10.1111/j.1399-302X.2007.00369.x

Ferreira, D.C., Paiva, S.M., Carmo, F.L., Rôças, I.N., Rosado, A.S., Santos, K. N., Siqueira, J.F., (2011). Identification of herpesviruses types 1 to 8 and human papillomavirus in acute apical abscesses. Journal Of Endodontics, 37(1), 10-16. https://doi.org/10.1016/j.joen.2010.09.009

Grenier, G.G., Gagnon, G.G., Grenier, D.D., (2009). Detection of herpetic viruses in gingival crevicular fluid of pa-tients suffering from periodontal diseases: prevalence and effect of treatment. Oral Microbiology & Immunology, 24(6), 506-509. https://doi.org/10.1111/j.1399-302X.2009.00542.x

Hernádi, K., Gyöngyösi, E., Mészáros, B., Szakács, L., Szalmás, A., Csoma, E., et ál. (2013). Elevated tumor necrosis factor-alpha expression in periapical lesions infected by epstein-barr virus. Journal Of Endodontics, 39(4), 456-460. https://doi.org/10.1016/j.joen.2012.12.028

Kakehashi, S., Stanley, H.R., Fitzgerald, R.J., (1965). The effects of surgical exposures of dental pulps in germ-free and conventional laboratory rats. Oral Surg Oral Med Oral Pathol, 20, 340-349. https://doi.org/10.1016/0030-4220(65)90166-0

Li, H., Chen, V., Chen, Y., Baumgartner, J.C., Machida, C.A., (2009). Herpesviruses in endodontic pathoses: associa-tion of epstein-barr virus with irreversible pulpitis and apical periodontitis. Journal of Endodontics, 35(1), 23-9. https://doi.org/10.1016/j.joen.2008.09.017

Lin, Y.L., & Li, M.M. (2009). Human cytomegalovirus and epstein–barr virus inhibit oral bacteria-induced ma-crophage activation and phagocytosis. Oral Microbiology & Immunology, 24(3), 243-248. doi:10.1111/j.1399-302X.2009.00504.x

Mandalunis, P.M., (2006). Remodelación ósea. Actualizaciones en osteología, 2(1), 16-18.

Sabeti, M., & Slots, J., (2004). Herpesviral-bacterial coinfection in periapical pathosis . Journal of Endodontics, 30(2), 69-72. https://doi.org/10.1097/00004770-200402000-00001

Sabeti, M., Kermani, V., Sabeti, S., Simon, J.H., (2012). Significance of human cytomegalovirus and epstein-barr virus in inducing cytokine expression in periapical lesions. Journal Of Endodontics, 38(1), 47-50. https://doi.org/10.1016/j.joen.2011.09.026

Sabeti, M., Simon, J.H., Slots, J., (2003b). Cytomegalovirus and epstein–barr virus are associated with symptomatic periapical pathosis. Oral Microbiology & Immunology, 18, 327–328. https://doi.org/10.1034/j.1399-302X.2003.00079.x

Sabeti, M., Valles, Y., Nowzari, H., Simon, J. H., Kermani-Arab, V., Slots, J., (2003c). Cytomegalovirus and epstein– barr virus DNA transcription in endodontic symptomatic lesions. Oral Microbiology & Immunology, 18(2), 104-108. https://doi.org/10.1034/j.1399-302X.2003.00055.x

Sabeti, M., Simon, J.H., Nowzari, H., Slots, J., (2003a). Cytomegalovirus and epstein-barr virus active infection in periapical lesions of teeth with intact crowns. Jorunal of Endodontics, 29(5), 321-323.

Sabeti, M.M., Daneshmand, A.A., Simon, J.H., Slots, J.J., (2009). Cytomegalovirus-infected inflammatory cells in dental periapical lesions. Oral Microbiology & Immunology, 24(5), 434-436. https://doi.org/10.1111/j.1399-302X.2009.00540.x

Slots J., Nowzari H., Sabeti M., (2004). Cytomegalovirus infection in symptomatic periapical pathosis. Internatio-nal Endodontic Journal, 37, 519–524. https://doi.org/10.1111/j.1365-2591.2004.00816.x

Stashenko, P. Teles, R., D’Souza, R., (1998). Periapical inflammatory responses and their modulation. Critical Re-views in Oral Biology & Medicine, 9(4), 498-521. https://doi.org/10.1177/10454411980090040701

Yazdi, K.A., Sabeti, M., Jabalameli, F., Emaneini, M., Kolahdouzan, S.A., Slots, J., (2008). Relationship between human cytomegalovirus transcription and symptomatic apical periodontitis in Iran. Oral Microbiology & Immu-nology, 23, 510–514. https://doi.org/10.1111/j.1399-302X.2008.00471.x

Yeung, R.S., (2004). The osteoprotegerin/osteoprotegerin ligand family: role in inflammation and bone loss. Jour-nal of Rheumatology, 31(5), 844-846.

Yildirim S, Yapar M, Kubar A, Slots J., (2006). Human cytomegalovirus, epstein-barr virus and bone resorption-inducing cytokines in periapical lesions of deciduous teeth. Oral Microbiology & Immunology, 21, 107–111. https://doi.org/10.1111/j.1399-302X.2006.00268.x

Descargas

Publicado

2015-01-01

Cómo citar

Herrera García, A., & Castro Mora, S. (2015). Influencia de los herpesvirus en la patogénesis periapical. O.V., 1(22), 68–73. https://doi.org/10.59334/ROV.v1i22.285

Número

Sección

Artículo de investigación

Métricas alternativas